The team Mr. Valls leads has developed genetic tools to stably transform R. solanacearum, which have been essential to study the infection process in resistant and susceptible plants, to evaluate plant germplasm for resistance to bacterial wilt and to study the plant and bacterial transcriptomes. The team has established the main bottlenecks in resistant tomato that restrict R. solanacearum infection and have determined that ligno-suberin vascular coatings and, more precisely, tyramine-derived hydroxycinnamic acid amides are involved in pathogen restriction. The team has also determined four genetic programmes that are deployed by R. solanacearum to colonise plants,that the bacterial type III Secretion system is expressed throughout infection and have identified some plant targets of bacterial effector proteins. He has also contributed to the work led by NS Coll to characterise the process of plant cell death and the role played by metacaspases in it.
Accomplishments
Our main goals are todecipher the genetic determinants of bacterial plant diseases and tocharacterize the main executioners of the hypersensitive response cell death.We combine genetics, biochemistry and molecular biology in an integratedapproach to study both plants and their pathogens.The Bacterial Plant Diseases and Cell Death team is located in the Centre forResearch in Agricultural Genomics (CRAG) in Bellaterra, Barcelona. The CRAG isa consortium between the Spanish National Research Council (CSIC), the CatalanInstitute for Food and Agricultural Research and Technology (IRTA), theUniversity of Barcelona (UB) and the Autonomous University of Barcelona (UAB).
For more info: http://www.ub.edu/bactplantdiseases/
Bacterial wilt caused by the wide-host range pathogen Ralstonia solanacearum is a devastating disease affecting over 200 plant species from disparate families including potato, tomato, peanut, eucalyptus, clove, etc. We study the expression and the functions of R. solanacearum virulence genes inside its plant hosts. The work includes generation of straind mutant or over expressing candidate genes and evaluating their impact on bacterial virulence and survival.
From June 9 to August 31, 2025 (adjustable at the discretion of the organisation)